Abfraction is a theoretical concept explaining a loss of tooth structure not caused by tooth decay (non-carious cervical lesions). It is suggested that these lesions. Multiple factors are seen as contributing to the development of noncarious cervical lesions (NCCLs). The term abfraction is applied to these lesions in relation to. Evidence supports that abfraction lesions, as any NCCLs, have a multifactorial etiology. Particularly, the cervical wear of abfraction can occur.
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J Oral Maxillofac Pathol.
Dietary erosion results due to high intake of foods or drinks having a variety of acids, from citrus fruits, juices citric acidsoft aerated drinks, wine and other carbonated drinks carbonic acid and other acids. Guidelines for the enhancement of esthetics may help the patient when selecting treatment options after they have recognized their goals.
NCCL, noncarious cervical lesion. If left untreated, abfraction can lead to root canal infections, severe decay, and even tooth loss. Clinical features of erosive lesions also include broad concavities within smooth tooth enamel, loss of enamel surface anatomy, increase in incisal translucency, and incisal chipping and cupping out of occlusal surfaces with dentine exposure.
Approaches to determine lesion activity include the use of standardized intra-oral photographs, study models, and measurement of lesion dimensions over time.
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The factors related to restorative retention are location of tooth, age of patient and occlusion [ 55 ]. The effects of occlusal loading on the margins of cervical restorations.
Guidelines Upcoming Special Issues. This reduces lesionz and restores the tooth structure. Abfraction may avfraction defined as V-shaped or wedge shaped defect at the cervical region of a tooth with different clinical appearances mostly seen as angular notch like depressions on the facial surface of tooth structure at the junction of tooth and gingiva, this may be due to flexure of the cusp leading to mechanical overloading and may also be accompanied by pathological wear such as regressive alterations of teeth.
The Class V lesion — aetiology and restoration. Effect of lateral excursive movements on the progression of abfraction lesions. This review focuses on the current knowledge and available treatment strategies for abfraction lesions. We can only repair the teeth. Salivary glands Benign lymphoepithelial lesion Ectopic salivary gland tissue Frey’s syndrome HIV salivary gland disease Necrotizing sialometaplasia Mucocele Ranula Pneumoparotitis Salivary duct stricture Salivary gland aplasia Salivary gland atresia Salivary gland diverticulum Salivary gland fistula Salivary gland hyperplasia Salivary gland hypoplasia Salivary gland neoplasms Benign: Abfractions lesions, as any other NCCLs, have a multifactorial etiology.
If there are concerns around aesthetics or clinical consequences such as dentinal hypersensitivitya dental restoration white filling may be a suitable treatment option. Making sense of sensitivity. In extreme cases of Hypersensitivity unresponsive to non-invasive procedures, a restoration might be required. An abfraction can cause pain and could potentially lead to tooth loss if left untreated.
Upon examination, shiny facets on the teeth or existing restorations may be indicators of the presence of erosive processes. In an era of personalized dentistry, patient risk factors for NCCLs must be identified and addressed before any treatment is performed. Other non-invasive treatments include temporary sealants such as Varnishes and Dentin bonding agents. Treatment of cervical sensitivity with a root sealant.
Abfraction lesions will generally occur in the region on the tooth where the greatest tensile stress is located. Dentinal hypersensitivity is a response to stimuli caused by short, sharp pain. In addition to a thorough medical history, which should include an evaluation for gastroesophageal reflux disease, eating disorders, and dietary contributors, one should evaluate occlusion, parafunction, and oral habits, including occupational and ritual behaviors.
Visit for more related articles at Dentistry. Regularly biting or chewing objects like ice, fingernails, or pen caps A misaligned bite Restorations that do not fit properly How Is an Abfraction Treated?
Retrieved from ” https: Guidelines for the enhancement of esthetics may help the patient when selecting treatment options after they have recognized their goals. Hunter-Schreger Band patterns in human tooth enamel. J Tenn Dent Assoc.
Abfraction lesions: etiology, diagnosis, and treatment options
Clinical assessment of non carious cervical lesion using swept-source optical coherence tomography. Troy Office In the combined restorative-surgical approach, the restoration qbfraction be placed prior to the surgical procedure for better visibility of the operative field and for the finished restoration to provide a stable, hard, and convex substrate for the coronally advanced flap CAF.
Particularly, the cervical wear of abfraction can occur as a result of normal and abnormal tooth function and may also be accompanied by pathological wear, such as abrasion and erosion. The term lesios was first published in in a journal article dedicated to distinguishing the lesion.
Other important abfraciton such as tooth rotation, extrusion, and a clinically nonidentifiable CEJ may also impact the degree of root coverage. Efficacy of periodontal plastic surgery procedures in the treatment of localized facial gingival recessions. The abfraction lesions like any other NCCLs have a multifactorial etiology. Bartlett DW, Shah P. The Class V lesion — aetiology and restoration. Other terms bio-corrosion has also been introduced to include all forms of biochemical and electrochemical degradation As seen in Figure 4 [ 9 ].
Marcy Goldin and Dr. Clinical effectiveness of contemporary adhesives for the restoration of non-carious cervical lesions. Clinical assessment of non carious cervical lesion using swept-source optical coherence tomography.
abfrcation The defense mechanisms activated in teeth as a result of wear include the formation of reactionary and reparative dentin and the obstruction of exposed dentinal tubules by mineral deposits. Zucchelli G, Mounssif I.